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In melanocytes of vitiligo patients, elevated ROS, membrane lipid peroxidation, altered cardiolipin distribution, and defects in the tricarboxylic acid cycle activate the release of cytochrome and caspase cascades, leading to macromolecular damage, mitochondrial dysfunction, release of molecular patterns, and changes in mitochondrial membrane potential (Lin et al., 2024)
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Biomaterials 2011, 32, 4943–4950
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